Sepsis is defined as an inappropriate inflammatory response syndrome with concomintant infection. The definitions are changing all the time, and thus it is difficult to make standardised questions – particularly ones that will not require immediate revision – so effort has been made to tailor this quiz to Kumar and Clark’s current guidelines – though note in 2016, the notions of Severe Sepsis were redefined. Watch this space for more changes! In the meantime, test your knowledge on some fundamental clinical scenarios of the septic patient.
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Introduction to Sepsis MCQ
An acquired infective state
Pyrexia, leuocytosis and tachycardia/pnoea
Oliguria and increased INR
Hypovolaemic state evidenced post-Hartmann bolus resuscitative attempts
Decreased respiratory rate
Principally increase chronotrophy and inotrophy of the cardiomyocyte
located in the vascular wall and heart
Cause vasodilaton of peripheral vessels
Mediate vasodilation of mesenteric, renal, coronary and cerebral circulation
As first line medication
In patients at risk of bradyarrhythmia
To decrease SVR
Only when the patient is haemodynamically stable
Human growth hormone (hGH)
As a first line medication
Concomitantly with dopamine
Concomitantly with vasopressin
In cases of ADH-resistant resuscitation
Radiala and carotid pulses
Tachyarrhythmic cardiogenic shock
Low volume sepsis
Stem 1 of 4The following is an "SBAR" handover from an Advanced Paramedic to the F1 on-call in ED
Mr Jones, 65YO male, found short of breath, warm and clammy to touch with a decreased LOC. He has stable angina and controlled type 2 diabetes. He has a normal BM, sats on oxygen of 95%, heart rate of 100 and resp rate of 27. His temperature is 38.5º and he is 100 SBP. I'm concerned the patient is septic and so began with high flow oxygen, broad spectrum antibiotic IV and IV bolus of Hartmann solution pre-hospitally.How many septic red flags does this patient have?
Stem 2 of 4The patient has a confirmed infection. What score does the patient get on qSOFA?
Stem 3 of 4Which of the following are you not taking from the patient for investigation immediately?
Stem 4 of 4How do you anticipate the sCreat to be in the patient?
Increased due to afferent arteriolar vasodilation
Decreased due to hypoxaemic acidaemia of organs
Increased due to efferent arteriolar vasoconstriction
Increased due to AKI
Osteological medulla ischaemia
Overload of the cardiovascular system
Perfusion failure of hepatocytes