This is yet another stem of questions using the laws of flow physiology to elucidate the means of heart failure and compensation for different volume-ion changes in disease states. Enjoy!
Reviewed by: awaiting review
Control of Cardiac Output
Increases adrenergic response
A decrease in preload
An increase in the contractility of the heart
An increase in afterload
A decrease in volume of the chamber
it utilises mechanoceptors
It transmits afferents to the medulla via the tenth cranial nerve
An increase in cholinergic efferents results
An increase in noradrenergic efferents results
Increase of venous compliance
An increase of cardioaccelerator nerve functionality
Increasing aortic arch pressure
Stem 1 of 4
A patient is recovering after a myocardial infarction. Under LaPlace and Starling's law his heart must necessarily work greater and the cardiologist cautions the patient is at risk of hypertrophic cardiomyopathy.Under Starling's law, what is EDV proportional to?
Stem 2 of 4In the short term, which of the below scenarios is the patient's myocardium less likely to undergo?
Increases cardioaccelerator sympathetic postganglionics
Increased myocardial contracture force
Remodelling of the heart chamber
Increase of the HR
Stem 3 of 4Sarcomere length, ergo Starling's law of contracture, is determined by ...
Stem 4 of 4The patient had an infarct to the artery determining coronary dominance during his MI. How would you classify this vessel functionally?
An increase in systemic vascular resistance
A decrease in myocardial contractility
A decrease in the capacitance vasomotor activity
An increase in the vasodilation of the caval circulation
Increase to CO increases EDV
Increase to CO decreases EDV
Decrease to CO increases EDV
Decrease to CO decreases EDV